Matthew Voas has extensive biological laboratory research experience spanning over 18 years. In addition to his doctoral work at M.I.T., he was a postdoctoral fellow at Stanford University, and a research faculty member at the University of Utah. His training is particular strong in the fields of molecular biology, genetics, and the development of the nervous system. He has authored numerous scientific publications, presented his work at scientific conferences and universities, and is the recipient of an individual National Research Service Award. In addition to his knowledge of the biological sciences, Dr. Voas’ training has imparted strong analytical skills that he applies to any scientific questions that may impact a client matter.
Dr. Voas joined Hughes, Hubbard & Reed in 2014, and was approved to practice before the United States Patent and Trademark Office in 2016. He has contributed to matters in several areas, including intellectual property, product liability, and regulatory compliance. He supports the work of counsel in numerous ways, such as by researching and developing scientific arguments, analyzing the scientific content of documents, and helping to identify, develop, and prepare expert witnesses. This work has supported clients involved in patent infringement litigation, IPR challenges before the USPTO, and FDA compliance.
Individual neuronal subtypes exhibit diversity in CNS myelination mediated by synaptic vesicle release (2016). Koudelka S., Voas M.G., Almeida R.G., Baraban M., Soetaert J., Meyer M.P., Talbot W.S., Lyons D.A. Current Biology, 26(11), 1447-1455.
Mutation of sec63 in zebrafish causes defects in myelinated axons and liver pathology (2013). Monk K.R., Voas M.G., Franzini-Armstrong C., Hakkinen I.S., Talbot W.S. Disease Models and Mechanisms, 6(1), 930-942.
DNA demethylase activity maintains intestinal cells in an undifferentiated state following loss of APC (2010). Rai K., Sarkar S., Broadbent T.J., Voas M., Grossmann K.F., Nadauld L.D., Dehghanizadeh S., Hagos F.T., Li Y., Toth R.K., Chidester S., Bahr T.M., Johnson W.E., Sklow B., Burt R., Cairns B.R., Jones D.A. Cell, 142(6), 930-942.
Alpha II-spectrin is essential for assembly of the nodes of Ranvier in myelinated axons (2007). Voas M.G., Lyons D.A., Naylor S.G., Arana N., Rasband M.N., Talbot W.S. Current Biology, 17(6), 562-568.
Nsf is essential for organization of myelinated axons in zebrafish (2006). Woods I.G., Lyons D.A., Voas M.G., Pogoda H.M., Talbot W.S. Current Biology, 16(7), 636-648.
The Drosophila nucleosome remodeling factor NURF is required for Ecdysteroid signaling and metamorphosis (2005). Badenhorst P., Xiao H., Cherbas L., Kwon S.Y., Voas M., Rebay I., Cherbas P., Wu C. Genes & Development, 19(21), 2540-2545.
Erbb3 and erbb2 are essential for schwann cell migration and myelination in zebrafish. Lyons D.A., Pogoda H.M., Voas M.G., Woods I.G., Diamond B., Nix R., Arana N., Jacobs J., Talbot W.S. Current Biology, 15(6), 513-524.
Signal integration during development: insights from the Drosophila eye (2004). Voas M.G., Rebay I. Developmental Dynamics, 229(1), 162-175.
The novel plant homeodomain protein rhinoceros antagonizes Ras signaling in the Drosophila eye (2003). Voas M.G., Rebay I. Genetics, 165(4), 1993-2006.
Biological functions of the ISWI chromatin remodeling complex NURF (2002). Badenhorst P., Voas M., Rebay I., Wu C. Genes & Development, 16(24), 3186-3198.
A genetic screen for novel components of the Ras/Mitogen-activated protein kinase signaling pathway that interact with the yan gene of Drosophila identifies split ends, a new RNA recognition motif-containing protein. Rebay I., Chen F., Hsiao F., Kolodziej P.A., Kuang B.H., Laverty T., Suh C., Voas M., Williams A., Rubin G.M. Genetics, 154(2), 695-712.